It’s long been known that a mutation in the BRAF gene causes moles to start growing, nonetheless it wasn’t understood why they stop growing. The BRAF mutation that stimulates the initial growth of moles also stimulates the creation of a tumor suppressor protein, p15, which ultimately works as a powerful brake on further cell division, research senior author Dr. Todd Ridky, an assistant professor of dermatology at the University of Pennsylvania, described in a university news release. It’s this cell division that ultimately allows the transition from a standard mole into melanoma. When mole cells lose the p15 brake, cells can begin dividing again and will progress into cancer, he explained.Neither of these ongoing celebrations endorse or suggest any commercial products, services, or equipment.

ACC: New community-wide collaboration to reduce impact of chronic diseases A new community-wide collaboration to reduce the impact of chronic empower and disease patients is generating impressive early benefits, leaders of the Accountable Care Community initiative said today. The Akron-based Austen BioInnovation Institute in Akron is leading the initiative using its founding institutional associates and more than 60 public and private community companions. The groundbreaking effort supported by the Centers for Disease Prevention and Control unites medical, public health and social technology professions, non-profits and faith-structured and community institutions for an ‘all-hands-on-deck’ approach to public health.